Quote from Dr. Garrett Smith on April 28, 2019, 7:17 pm

If this is happening to cheetahs in zoos under the watch of zoologists and zookeepers, do you STILL think it isn't happening in people?

I will be interjecting below.

Veno-occlusive Disease of the Liver in Captive Cheetah

Abstract. Liver tissues from 126 captive cheetah were evaluated by light microscopy and histochemistry; eight animals were evaluated by electron microscopy. The main hepatic lesion, a vascular lesion resembling veno-occlusive disease (VOD) of the liver and characterized by subendothelial fibrosis and proliferation of smooth muscle-like cells in the central veins, was seen in 60% of the sexually mature cheetah.

When something is this prevalent (60%), it's likely due to a poison/toxin.

Veno-Occlusive Disease (VOD) - "A condition in which some of the veins in the liver are blocked. This causes a decrease in blood flow inside the liver and may lead to liver damage." 

Although this hepatic vascular lesion was seen in cheetah as young as 1 year of age, the most severe lesions, usually associated with liver failure, were found in cheetah between the ages of 6 and 11.

Poison/"Vitamin A" toxicity is a disease of ACCUMULATION, so it only makes sense that it would be seen more in the older/mature cheetahs.  Yes, it is also happening to the young cheetahs, and is happening to children every day.

There was no sex predisposition, and in approximately 40% of the VOD cases, liver disease was not suspected clinically or at necropsy.

Note that the liver disease being caused by Poison/"Vitamin A" was often not suspected in the animals when they were ALIVE or even after they were DEAD!!!

"But there's no way I have this problem"...famous last words of those in denial about the Poison/"Vitamin A" epidemic.

VOD was found in other felidae, especially in the snow leopard. High levels of vitamin A in livers, as well as in diets of the cheetah, could be a contributing factor in the development of VOD in some groups of cheetah.

Liver biopsy is the GOLD STANDARD of diagnosing Poison/"Vitamin A" toxicity.  They found high levels in the cheetah livers, AND in their diets.  Not a coincidence!

Dietary factors can be suspected in the etiology of a non-infectious disease with high incidence in groups of animals from a wide geographical range and across felid species boundaries.6,24,29,32,40,41 Cheetah in the wild prey mostly on small antelope,* while those kept in North American zoos are fed commercially prepared feline diets, sometimes supplemented with raw liver, or zoo prepared diets. 

It's their diet.  Raw liver, what is that high in again?  Oh yes, Poison/"Vitamin A"!!!

The specific cause for VOD in each individual animal cannot be identified. However, based on correlations between morphologic findings and dietary analyses, it is suggested that at least one dietary factor may, at the present time, play a role in the pathogenesis of hepatic vascular lesions. Hypervitaminosis A may be responsible mainly for Ito cell proliferation and peri-sinusoidal fibrosis, which could progress to central vein sclerosis, as has already been demonstrated experimentally in the rat.20 Vitamin A excess in cheetah could also increase the severity of the lesion and accelerate the clinical manifestation of VOD from other causes. By simple manipulation of the diet, this factor can be corrected, allowing future opportunity to identify other possible factors that are not obvious at this time.

They've now matched up this problem among rats, cheetahs, and snow leopards.

"By simple manipulation of the diet, this factor can be corrected..."

We call it the Poison/"Vitamin A" and Glyphosate Detox Program.