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Schizophrenia - "Three independent lines of evidence suggest retinoids as causal to schizophrenia"

These scientists have established that it is Poison/"Vitamin A" that is the main cause of schizophrenia.  These are the juiciest excerpts, there's more at the link.

Three independent lines of evidence suggest retinoids as causal to schizophrenia

RETINOID-RELATED ANOMALIES IN SCHIZOPHRENIC PEDIGREES
The first line of evidence firmly connecting retinoids to schizophrenia is that retinoid toxicity or deficit has repeatedly been shown to result in symptom presentations that, though more severe in extent, resemble the stigmata of schizophrenia, e.g., thought disorder, mental deficit, enlarged ventricles, agenesis of the corpus callosum, microcephaly, and a variety of major and minor congenital malformations, among which craniofacial and digital anomalies are prominent.
[...]
CONVERGENCE OF RETINOID LOCI AND SCHIZOPHRENIA LOCI (CONVERGENT LOCI)
Genetic linkage data now support the evidence from family history, twin, adoption (13), and epidemiologic studies (14) that schizophrenia is a complex non-Mendelian illness with a strong genetic component. A second line of evidence implicating retinoids in the genetic etiology of schizophrenia comes from studies that have identified specific replicable loci that have been suggestively linked to schizophrenia (13). The common factor among these promising schizophrenia loci is that they are known loci of genes within the retinoid signaling system or metabolic cascade.
[...]
EXPRESSION OF SCHIZOPHRENIA CANDIDATE GENES IS REGULATED BY RETINOIC ACID
The third line of evidence relates the targets of retinoid regulation to numerous schizophrenia candidate genes. Retinoic acid regulates target genes by combining with RARs and RXRs, which control transcription of these targets. After activation in the presence of retinoic acid, these receptors bind to specific DNA sequences within the promoter–enhancer regions of target genes. These DNA motifs are known as retinoic acid response elements (RAREs or RXREs). Through this mechanism retinoic acid receptors and their partners regulate the expression of multiple target genes. The regulation in the expression of these target genes results in changes in cell function that produces normal or abnormal physiological states (15).

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