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WAPF study shows cod liver oil increases postpartum hemorrhaging, Poison/"Vitamin A" and oral Vitamin D increases hemorrhaging (bleeding)

One day, I was looking up the (free) fatty acid profile of beef tallow, and my internet searching brought me to this very interesting link.  As some say, nothing happens by accident.  I am going to save the screenshot here as an attachment so that this won't disappear from the interwebz.

The Weston A. Price Foundation did an observational study...a VERY interesting correlation was found.

Cod Liver Oil Survey – Preliminary Results

Fatty Acid Analysis of Grass-fed and Grain-fed Beef Tallow

"In April of 2012, we received an anecdotal report from a midwife of several women experiencing severe postpartum hemorrhages while reportedly following the dietary recommendations of the Weston A. Price Foundation. Concerned that the large amount of omega-3 fatty acids found in cod liver oil could have contributed to the hemorrhages through their blood-thinning properties, especially if not balanced by adequate liver, egg yolks, and other sources of arachidonic acid, we conducted a survey to determine whether postpartum hemorrhage and vaginal bleeding during pregnancy were associated with the use of cod liver oil or the dietary recommendations of the foundation. To reduce the risk of bias and increase the amount of information that could be gained from the survey, we circulated the survey widely on the Internet and asked about a large number of foods, perinatal complications, and medications. Over 3,500 women following many different diets completed the survey.

There was no association between the type of diet the women reported following and any of the complications or medications. Women who reported taking cod liver oil were 30 percent more likely to experience postpartum hemorrhage, but the difference was not statistically significant (P=0.09), meaning there is a reasonable likelihood the association could be due to chance. Several observations suggest this is unlikely to be a true biological effect: there was no association with the dose of cod liver oil; omega-3 fatty acids are also found in fish oil, but there was no association with the use of fish oil; there was no association between cod liver oil and the risk of vaginal bleeding during pregnancy; and there was no association between cod liver oil and the use of medications used to control bleeding. By contrast, cod liver oil was associated with a large (63 percent) and statistically significant (P<0.001) drop in the risk of preeclampsia, and the magnitude of the drop in risk correlated well with the dose of cod liver oil (P<0.001). Since this is an observational study, it cannot demonstrate cause-and-effect relationships, but this association could reflect a protective effect of the fat-soluble vitamins in cod liver oil.

The data gathered from this survey are voluminous and will be reported in much greater detail in the next issue of this journal.

This article appeared in Wise Traditions in Food, Farming and the Healing Arts, the quarterly journal of the Weston A. Price Foundation, Winter 2013."

The screenshot for posterity is attached.  My interpretation of the above:

  • First and most importantly, I am of the opinion that this study was biased from the beginning. I assume that they undertook this study with the intention of demonstrating that there WEREN'T problems with their dietary recommendations. Companies protect their interests, right?  I mean, who trusts companies to properly investigate themselves and find wrongdoing?  Do you really trust Monsanto/Bayer to honestly study and report on glyphosate/Roundup?  Why would we assume any other company is different?
  • Women who took cod liver oil were 30% more likely to experience postpartum hemorrhage(!).  Then we have the "but it wasn't statistically significant".  Honestly, I'm surprised this was even reported, being that it is counter to the WAPF's narrative on eating tons of Poison/"Vitamin A" and oral sources of Vitamin D.  As for the lack of statistical significance part, there are three types of lies:  "Lies, damn lies, and statistics."  I consider 30% to be VERY significant, regardless of the statistical dance.
  • There was NO association between specific doses of cod liver oil and postpartum hemorrhage.  What?!?!  So ANY/EVERY dose that was taken possibly increased the risk?  That sounds pretty scary to me.
  • There was NO association for fish oil and postpartum hemorrhage.  Interesting.  What are the two main differences between cod liver oil (that comes from cod--a fish--obviously) and fish oil?  A high content of Poison/"Vitamin A" and Vitamin D3.  So, the cause of the problems from cod liver oil are quite likely from something it contains that fish oil does not (fish oil may contain some Poison/"Vitamin A", this is true, but it is magnitudes less than that in cod liver oil). This fact gives us some primary suspects!
  • Intake of Poison/"Vitamin A" and oral sources of Vitamin D (a hormone therapy that in my opinion calcifies people to a slow death) was not assessed.  Hard to find a problem that is not looked for.

That last bullet point is very important to the connections I'm about to make.  BOTH hypervitaminosis A and hypervitaminosis D have been connected to hemorrhage in the literature.  They are both in cod liver oil in very significant amounts.

First, Poison/"Vitamin A".  There is tons of evidence in animals of this connection.

Hypervitaminosis A in the dog.
"Pathologic changes were degenerative epiphyseal plate, hemorrhage and exostotic proliferation of periosteum, fatty liver, and microcalculi in kidney."

The effect of hypervitaminosis A and other dietary factors on the young pig (also attached)
"Following reports of arctic explorers who "became ill after the ingestion of polar bear liver, Rodahl and Moore (1943) analyzed samples of seal and polar bear liver for vitamin A and found them to contain from 13,000 to 18,000 International Units (lU) of the vitamin per gram of wet weight. When rats were given approximately one gram of this liver dally, they became anemic, were paralyzed in the rear legs, and hemorrhaged under the skin and in the pericardium.Evidence for a malfunctioning blood-clotting mechanism was found. Not all animals were affected, however. These characteristics are typical of most reports on vitamin A toxicity.
Maddock et aJ. (1948) found high levels of vitamin A fed to rats resulted in hemorrhages in the subdural space, epididymus, skeletal muscles, and in the body cavities. The lymph nodes were hemorrhagic in appearance because their sinuses were filled with blood.

Rodahl (1950) observed the following effects due to hypervitaminosis A in the rat; pelt deterioration, muscular weakness, lack of appetite, reduced growth, soreness and bleeding of skin, alopecia, swelling of palpebrae, exophthalmos, stiffness of limbs, spontaneous fractures, hemorrhages, proteinuria, hematuria, diarrhea, intestinal bleeding, and anemia. No significant changes were found in blood calcium, phosphorus, potassium, total base, urea, or sugar. Bone ash was reduced, but bone ash calcium or phosphorus was not affected. Histologically, the usual findings were renal tubule degeneration, deposits of sudanophil droplets in the liver and in the adrenal cortex as well as general internal hemorrhage.
Adult pregnant rats given 25,000 to 50.000 lU of vitamin A per day by Moore and Wang (1943) developed fatal uterine hemorrhages or subpericardial hemorrhages. Subcutaneous and intramuscular hemorrhages were also found. These authors also remarked that there seemed to be a superficial resemblance to vitamin C deficiency in these rats.
Moore and Wang (1945) used pure crystalline vitamin A to produce toxicity in rats and found that one milligram of calciferol
[Vitamin D supplement] per day had no suppressive action on the incidence of bone fractures or hemorrhages resulting from the vitamin A.

Walker et (1947) found that massive doses of vitamin C to rats did not prevent bone fractures or hemorrhages due to hypervitaminosis A, but vitamin K diminished the incidence of hemorrhage although it had no effect on bone fractures. Vitamin K was partially effective in increasing hematocrit values which were depressed by hypervitaminosis A.
Hemorrhage in developing amphibia suffering from hypervitaminosis A was prevented by administration of epsilonaminocaproic acid by Weissmann et al. (1963a).
The first obvious indications that the pigs were being affected by the high level of vitamin A, other than sudden death, were a general unthrifty appearance, and a reluctance to move about the pen. Shortly thereafter some of the animals lost their ability to use their rear legs. Later, strength and control of the front limbs was also greatly diminished. Growth ceased, and generally weight losses were incurred. There was considerable lacrimation. Joints became swollen, especially in the rear legs. Hair coats were ragged in appearance and the skin, especially over the legs and abdomen, became dry and scaly. The skin around the top and between the hooves was cracked, and in some instances, bleeding occurred from these sites. Marked petechial hemorrhages appeared subcutaneously over the limbs and abdomen. Frequently the urine and feces contained blood.

Postmortem examination of the three pigs which died revealed that hemorrhages into several limb joints had occurred. In the intestine, subserosal and mucosal hemorrhages were evident. Subpericardial hemorrhage was found. Costochondral junctions were enlarged and, when cross-sectioned, the metaphyseal region was white and had the appearance of being decalcified. Histologically, the cartilage columns were disorganized. In one case a ruptured kidney with resulting hemorrhage into the peritoneal cavity was observed. Hemorrhages were noted in the area of the cortico-medullary junction of both kidneys of this animal.

Pigs other than those receiving 500,000 lU per pound of diet exhibited no symptoms of hypervitaminosis A at any time. When metatarsal bones were removed for analysis, it was evident that the blood clotting mechanism in those pigs on the highest level of vitamin A was defective."

Moving on to Vitamin D causing hemorrhaging:

Clinicopathological Studies on Vitamin D3 Toxicity and Therapeutic Evaluation of Aloe vera in Rats
"The rats died due to vitamin D3 toxicity (groups A and B) or sacrificed showing signs of vitamin D3 toxicity had severe emaciation and ruffled body coat. The heart was felt hard with white chalky deposits on epicardial surface. The stomach and intestines showed diffused white chalky deposits on serosal surface. The stomach and intestines revealed bloody ingesta in the lumen with marked hemorrhages on mucosa.
Hemorrhagic gastritis was also observed in some rats.
Other histopathological changes in lungs included marked hemorrhage, emphysema, and edema."

A case study of a dog getting poisoned by Vitamin D by eating rat bait (on that note, did you know that they KILL RATS by giving them massive doses of Vitamin D in RAT BAIT?):
"HISTORY:  This dog ingested 30 grams of mouse - rat bait; 36 hours later the dog was depressed and anorectic.  His condition continually deteriorated and the dog died of cardiac asystole approximately 72 hours after ingestion of the bait.

ETIOLOGIC DIAGNOSIS:  Renal and pulmonary hypervitaminosis D

SYNONYMS: Vitamin D nephropathy; vitamin D toxicity; vitamin D poisoning (intoxication)

Lung, bronchi, bronchioles and alveoli: Mineralization, multifocal, severe, with necrosis, hemorrhage, fibrin, and edema.

TYPICAL GROSS LESIONS:  Acute, high dose exposure: Gastric, small intestinal and myocardial hemorrhage

TYPICAL LIGHT MICROSCOPIC FINDINGS: Acute: Stomach-mucosal hemorrhage, crypt necrosis; heart- myocardial necrosis"

For those with the eyes to see and the brain to think, I would say that there is a definitely plausible connection between:

  1. OBSERVATIONS of increased postpartum hemorrhage in users of cod liver oil
  2. The FACT that cod liver oil is mainly used as a dietary supplement of Poison/"Vitamin A" & Vitamin D3
  3. The KNOWN hemorrhagic responses of mammals that ingest either or both of these two compounds in excess.

So if a woman was going through a "physically traumatic event" like giving birth, and they had been taking something during their pregnancy that is known to negatively affect blood clotting mechanisms in 3 separate ways (Poison/"Vitamin A", oral Vitamin D, and the decreased clotting associated with fish oil intake), it should make very logical sense that they would be at higher risk of postpartum hemorrhage.

Cod liver oil...not even once.  Make sure to share this with all the pregnant or soon-to-be pregnant ladies you know.




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Dr. Garrett Smith, the "Nutrition Detective"
Licensed Naturopathic Physician (NMD) in Arizona, home of the Love Your Liver program
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